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Furthermore, redox alterations have bidirectional role in developing insulin resistance: H 2 O 2 has been shown in mice to attenuate insulin resistance while chronic ROS production via mitochondria and NOXes contribute to development of insulin resistance by pro-inflammatory cytokines (Loh et al

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For example, while glutamate excitotoxicity increases oxidative stress and activates NRF2 signaling, it is often insufficient to prevent cell death (Xin et al., 2019